Anti-MTSS1 / MIM / Missing in metastasis

Item number Size Datasheet Manual SDS Delivery time Quantity Price
NSJ-RQ7190 100 µg - -

3 - 10 business days*

790.00€
 
0.5mg/ml if reconstituted with 0.2ml sterile DI water. Metastasis suppressor protein 1, also... more
Product information "Anti-MTSS1 / MIM / Missing in metastasis"
0.5mg/ml if reconstituted with 0.2ml sterile DI water. Metastasis suppressor protein 1, also called MIM or Missing in metastasis, is a protein that in humans is encoded by the MTSS1 gene. Enables actin monomer binding activity, identical protein binding activity, and signaling receptor binding activity. Predicted to be involved in cellular response to fluid shear stress, negative regulation of epithelial cell proliferation, and urogenital system development. Predicted to act upstream of or within several processes, including actin filament polymerization, adherens junction maintenance, and magnesium ion homeostasis. Located in actin cytoskeleton. Protein function: May be related to cancer progression or tumor metastasis in a variety of organ sites, most likely through an interaction with the actin cytoskeleton. [The UniProt Consortium]
Keywords: Anti-KIAA0429, Anti-Protein MTSS 1, Anti-Metastasis suppressor YGL-1, Anti-Missing in metastasis protein, Anti-Metastasis suppressor protein 1, MTSS1 Antibody / MIM / Missing in metastasis
Supplier: NSJ Bioreagents
Supplier-Nr: RQ7190

Properties

Application: WB, IHC (paraffin), FC, Direct ELISA
Antibody Type: Polyclonal
Conjugate: No
Host: Rabbit
Species reactivity: human, mouse, rat
Immunogen: Recombinant human protein (amino acids Q17-E692)
Format: Adhesion & Structure

Handling & Safety

Storage: +4°C
Shipping: +4°C (International: +4°C)
Caution
Our products are for laboratory research use only: Not for administration to humans!
You will get a certificate here
or to request a certificate of analysis.
Read, write and discuss reviews... more
Customer review for "Anti-MTSS1 / MIM / Missing in metastasis"
Write a review
or to review a product.
Viewed